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fent

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Quick breakdown on some compounds to grow taller
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Hope you enjoyed this thread, alot of it is slightly changed from heightmaxxing 202 on .gg with 1x new addition, Other then dropping some new height info soon
 

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fent said:
Quick breakdown on some compounds to grow taller
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Hope you enjoyed this thread, alot of it is slightly changed from heightmaxxing 202 on .gg with 1x new addition, Other then dropping some new height info soon
Click to expand...
good thread
 
fent said:
Onto our next topic, Aromatase inhibitors or AI for short.
The aromatase enzyme's main job is to convert androgens (test, deca or whatever) through a process known as aromatization to types of estrogens (estradiol, estrone). This is the main driver of growth plate closure.

Types of aromatase inhibitors
Letrozole: Im sure you've heard of it, it is the most popular letrozole is a Type 2, reversable, suicidal aromatase inhibitor, it is incredibly potent and inhibits 98-99% of circulating e2 at a dosage of 2.5mg
Pros: High e2 supression and slows bone maturation more then any other, increases natural test levels.
Cons: Lowers circulating IGF-1, Non suicidal, potential weaker bone mineral density, joint pain, e2 rebound if not careful

Anastrozole: A reversal non suicidal inhibitor and inhibits e2 at around 80-90% at a dosage of 1mg
Pro: Main pro is that it wil be easier on the body due to a lighter supression,
Cons: The same as letro pretty much (keeps igf-1 intact however) , e2 rebound if not careful

Aromasin: Type 1 suicidal inhibitor. Instead of reversibly blocking the aromatase enzyme, aromasin binds permanently to it, rendering the enzyme inactive for the rest of its lifespan which is also known as suicidal inhibition. Supresses e2 roughly 35-62% at a 25mg dose

Pros: No e2 rebound, little to no impact on igf-1, raises both free and total test
Cons: Pretty much the same nonetheless, joint pain and bone mineral density depletion also low supression of e2
Click to expand...
Unfortunetly, you can't inhibit any local estradiol found in your growth plates. It goes through a process called de novo synthesis and your body makes e2 from cholesterol.

The only way to inhibit local e2 (as far as i know) is to utilize DHT esters (eg: DHT Prop / DHT Enanthate) because it upregulates 5ar expression. Run a testosterone base (100-125mg) alongside with estered DHT, due to the 5ar upregulated expression more testosterone will convert into DHT directly in the growth plates, and its water info that DHT is anti estrogenic through 4 distinct mechanisms:
https://pmc.ncbi.nlm.nih.gov/articles/PMC425420/ (ER antagonism)

AR displaces ERa from DNA:

Research Resource: Interplay between the Genomic and Transcriptional Networks of Androgen Receptor and Estrogen Receptor α in Luminal Breast Cancer Cells - PMC

The cellular response to circulating sex steroids is more than the sum of individual hormone actions, instead representing an interplay between activities of the evolutionarily related steroid hormone receptors. An example of this interaction is in ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
pubmed.ncbi.nlm.nih.gov

Androgen receptor inhibits estrogen receptor-alpha activity and is prognostic in breast cancer - PubMed

There is emerging evidence that the balance between estrogen receptor-alpha (ER(alpha)) and androgen receptor (AR) signaling is a critical determinant of growth in the normal and malignant breast. In this study, we assessed AR status in a cohort of 215 invasive ductal breast carcinomas. AR and...
pubmed.ncbi.nlm.nih.gov pubmed.ncbi.nlm.nih.gov

AIB1 sequestration from DHT-activated AR:

AIB1 sequestration by androgen receptor inhibits estrogen-dependent cyclin D1 expression in breast cancer cells - PMC

Androgens, through their own receptor, play a protective role on breast tumor development and progression and counterbalance estrogen-dependent growth stimuli which are intimately linked to breast carcinogenesis. Cell counting by trypan blu ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov

Now the conclusion is that running aromatisable androgens in puberty is terrible even with using AIs/SERDs/SERMs, now running AIs (or anything that interacts wih estrogden) is debatable and can potentially make you taller. (not really my expertise in pharmacology cause im already insanely tall)

You should look into ERb agonists like WAY200070
 
fent said:
Quick breakdown on some compounds to grow taller
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Hope you enjoyed this thread, alot of it is slightly changed from heightmaxxing 202 on .gg with 1x new addition, Other then dropping some new height info soon
Click to expand...
don't post .gg slope u posted here tbh
 
Administrator said:
Unfortunetly, you can't inhibit any local estradiol found in your growth plates. It goes through a process called de novo synthesis and your body makes e2 from cholesterol.

The only way to inhibit local e2 (as far as i know) is to utilize DHT esters (eg: DHT Prop / DHT Enanthate) because it upregulates 5ar expression. Run a testosterone base (100-125mg) alongside with estered DHT, due to the 5ar upregulated expression more testosterone will convert into DHT directly in the growth plates, and its water info that DHT is anti estrogenic through 4 distinct mechanisms:
https://pmc.ncbi.nlm.nih.gov/articles/PMC425420/ (ER antagonism)

AR displaces ERa from DNA:

Research Resource: Interplay between the Genomic and Transcriptional Networks of Androgen Receptor and Estrogen Receptor α in Luminal Breast Cancer Cells - PMC

The cellular response to circulating sex steroids is more than the sum of individual hormone actions, instead representing an interplay between activities of the evolutionarily related steroid hormone receptors. An example of this interaction is in ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
pubmed.ncbi.nlm.nih.gov

Androgen receptor inhibits estrogen receptor-alpha activity and is prognostic in breast cancer - PubMed

There is emerging evidence that the balance between estrogen receptor-alpha (ER(alpha)) and androgen receptor (AR) signaling is a critical determinant of growth in the normal and malignant breast. In this study, we assessed AR status in a cohort of 215 invasive ductal breast carcinomas. AR and...
pubmed.ncbi.nlm.nih.gov pubmed.ncbi.nlm.nih.gov

AIB1 sequestration from DHT-activated AR:

AIB1 sequestration by androgen receptor inhibits estrogen-dependent cyclin D1 expression in breast cancer cells - PMC

Androgens, through their own receptor, play a protective role on breast tumor development and progression and counterbalance estrogen-dependent growth stimuli which are intimately linked to breast carcinogenesis. Cell counting by trypan blu ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov

Now the conclusion is that running aromatisable androgens in puberty is terrible even with using AIs/SERDs/SERMs, now running AIs (or anything that interacts wih estrogden) is debatable and can potentially make you taller. (not really my expertise in pharmacology cause im already insanely tall)

You should look into ERb agonists like WAY200070
Click to expand...
This isnt true, you can fully degrade ERa with SERD's
 
Administrator said:
Unfortunetly, you can't inhibit any local estradiol found in your growth plates. It goes through a process called de novo synthesis and your body makes e2 from cholesterol.

The only way to inhibit local e2 (as far as i know) is to utilize DHT esters (eg: DHT Prop / DHT Enanthate) because it upregulates 5ar expression. Run a testosterone base (100-125mg) alongside with estered DHT, due to the 5ar upregulated expression more testosterone will convert into DHT directly in the growth plates, and its water info that DHT is anti estrogenic through 4 distinct mechanisms:
https://pmc.ncbi.nlm.nih.gov/articles/PMC425420/ (ER antagonism)

AR displaces ERa from DNA:

Research Resource: Interplay between the Genomic and Transcriptional Networks of Androgen Receptor and Estrogen Receptor α in Luminal Breast Cancer Cells - PMC

The cellular response to circulating sex steroids is more than the sum of individual hormone actions, instead representing an interplay between activities of the evolutionarily related steroid hormone receptors. An example of this interaction is in ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
pubmed.ncbi.nlm.nih.gov

Androgen receptor inhibits estrogen receptor-alpha activity and is prognostic in breast cancer - PubMed

There is emerging evidence that the balance between estrogen receptor-alpha (ER(alpha)) and androgen receptor (AR) signaling is a critical determinant of growth in the normal and malignant breast. In this study, we assessed AR status in a cohort of 215 invasive ductal breast carcinomas. AR and...
pubmed.ncbi.nlm.nih.gov pubmed.ncbi.nlm.nih.gov

AIB1 sequestration from DHT-activated AR:

AIB1 sequestration by androgen receptor inhibits estrogen-dependent cyclin D1 expression in breast cancer cells - PMC

Androgens, through their own receptor, play a protective role on breast tumor development and progression and counterbalance estrogen-dependent growth stimuli which are intimately linked to breast carcinogenesis. Cell counting by trypan blu ...
pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov

Now the conclusion is that running aromatisable androgens in puberty is terrible even with using AIs/SERDs/SERMs, now running AIs (or anything that interacts wih estrogden) is debatable and can potentially make you taller. (not really my expertise in pharmacology cause im already insanely tall)

You should look into ERb agonists like WAY200070
Click to expand...
obviously you cant nuke ALL e2 but you can get rid of most of it with an ai SERM's and SERD's
 
Kaligula said:
Yeye those were the days .gg was actually good
Click to expand...
*muHHH KalLLigulAA sponsERed THreaddd bruVVV*
posts a botb from .org with a shittier format

bro icl that nigga was annoying asf even tho he was raging
 
Brian said:
*muHHH KalLLigulAA sponsERed THreaddd bruVVV*
posts a botb from .org with a shittier format

bro icl that nigga was annoying asf even tho he was raging
Click to expand...
Zynx/idkmanimao x Proex/Kaligula combo was so good now that i remember this.

We used to post such peak on both .org and than .gg for some time

Rly such good times back then
 
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